MSC to adipocyte (mouse): Difference between revisions
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|timepoint_design=Early focus | |timepoint_design=Early focus | ||
|tissue_cell_type=Mesenchymal>>adipose | |tissue_cell_type=Mesenchymal>>adipose | ||
|zenbu_config=http://fantom.gsc.riken.jp/zenbu/gLyphs/#config= | |zenbu_config=http://fantom.gsc.riken.jp/zenbu/gLyphs/#config=bIgXKE9JM1ml8VD8z8Sqc;loc=mm9::chr11:52039190..52143552+ | ||
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Revision as of 16:12, 12 February 2015
Series: | IN_VITRO DIFFERENTIATION SERIES |
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Species: | Mouse (Mus musculus) |
Genomic View: | Zenbu |
Expression table: | FILE |
Link to TET: | TET |
Sample providers : | Yasushi Okazaki |
Germ layer: | mesoderm |
Primary cells or cell line: | primary cells |
Time span: | 6 days |
Number of time points: | 16 |
CollapseOverview |
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Senile osteoporosis is the most common metabolic bone disease. This disease is often accompanied by increasing adipocytes in bone marrow tissues [1]. The ectopic adipocytes differentiation following bone loss seems to be caused by unbalanced differentiation of mesenchymal stem cells (MSCs) [2]. Although several differentiation regulators of MSCs have already been reported, little is known about the regulatory dynamics of bi-directional adipocytes/osteoblasts differentiation. |
ExpandSample description |
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ExpandQuality control |
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Profiled time course samples
Only samples that passed quality controls (Arner et al. 2015) are shown here. The entire set of samples are downloadable from FANTOM5 human / mouse samples